Interrupting Crystal to Calculus Conversion: The Future of Research in Urolithiasis
Author:
Publisher
Springer Nature Singapore
Link
https://link.springer.com/content/pdf/10.1007/978-981-16-8845-4_15
Reference44 articles.
1. Aggarwal KP, Narula S, Kakkar M, Tandon C (2013) Nephrolithiasis: molecular mechanism of renal stone formation and the critical role played by modulators. Biomed Res Int 2013:292953. https://doi.org/10.1155/2013/292953
2. Alamani BG, Rimer JD (2017) Molecular modifiers of kidney stones. Curr Opin Nephrol Hypertens 26(4):256–265. https://doi.org/10.1097/MNH.0000000000000330
3. Alelign T, Petros B (2018) Kidney stone disease: an update on current concepts. Adv Urol 2018:3068365. https://doi.org/10.1155/2018/3068365
4. Asplin JR, Mandel NS, Coe FL (1996) Evidence of calcium phosphate supersaturation in the loop of Henle. Am J Physiol 270(4 Pt 2):F604–F613. https://doi.org/10.1152/ajprenal.1996.270.4.F604
5. Baggio B, Gambaro G, Ossi E, Favaro S, Borsatti A (1983) Increased urinary excretion of renal enzymes in idiopathic calcium oxalate nephrolithiasis. J Urol 129(6):1161–1162. https://doi.org/10.1016/S0022-5347(17)52619-1
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