Abstract
AbstractIn 1995, Yoshida et al. proposed first the concept of “autoimmune pancreatitis” (AIP). Since then, AIP has been accepted as a new pancreatic inflammatory disease and is now divided two subtypes. Type 1 AIP affected immunoglobulin G4 (IgG4) and implicates the pancreatic manifestation of IgG4-related disease, while type 2 is characterized by neutrophil infiltration and granulocytic epithelial lesions (GEL). Recent research has clarified the clinical and pathophysiological aspects of type 1 AIP, which is more than type 2 among the Japanese population. However, many details remain unclear about the pathogenesis and progression of this disease. In this review, we discuss the current knowledge and recent advances relating to type 1 AIP.
Funder
Japan Society for the Promotion of Science
Japan Science Society
Ministry of Health, Labour and Welfare
Japan Agency for Medical Research and Development
The branding program as a world-leading research university on intractable immune and allergic diseases supported by the Ministry of Education, Culture, Sports, Science and Technology of Japan.
grants-in-aid from the Ministry of Education, Culture, Sports, Science and Technology of Japan
Publisher
Springer Science and Business Media LLC
Cited by
4 articles.
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