Defects of TGF-β receptor signaling in mammary cell tumorigenesis

Author:

Brattain Michael G.,Ko Yong,Banerji Sunandita S.,Wu Gengfei,Willson James K. V.

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology

Reference46 articles.

1. N. M. Hoosein, M. K. McKnight, A. E. Levine, K. M. Mulder, K. E. Childress, D. E. Brattain, and M. G. Brattain (1989). Differential sensitivities of subclasses of human colon carcinoma cell lines to the growth inhibitory effects of TGF-β1.Exp. Cell Res. 181:442–453.

2. A. M. Manning, A. C. Williams, S. M. Gurne, and C. Paraskeva (1991). Differential sensitivity of human colonic adenoma and carcinoma cells to TGF-β1. Conversion of an adenoma cell line to a tumorigenic phenotype is accompanied by a reduced response to inhibiting affects of TGF-β.Oncogene 6:1471–1476.

3. S. Markowitz, L. Meyeroff, M. Cooper, J. Trascoff, M. Kochera, J. Lutterbaugh, M. Swiriduk, and J. K. V. Willson (1994). A benign cultured colon adenoma bears 3 genetically altered oncogenes, but progresses to tumorigenicity and TGF-β independence without inactivating the p53 tumor suppressor gene.J. Clin. Invest. 93:1005–1013.

4. C. L. Arteaga, A. K. Tandon, D. D. VonHoff, and C. K. Osborne (1988). TGF-β: Potential autocrine growth control inhibitor of estrogen receptor negative human breast cancer cells.Cancer Res. 48:3898–3904.

5. C. L. Arteaga, T. Carty-Dugger, H. C. Moses, S. D. Hurd, and J. A. Pietenpol (1993). TGF-β1 can induce estrogen independent tumorigenicity of human breast cancer cells in athymic mice.Cell Growth Differ. 4:193–201.

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