Effects of histone acetyltransferase (HAT) and histone deacetylase (HDAC) inhibitors on proliferative, differentiative, and regenerative functions of Toll-like receptor 2 (TLR-2)-stimulated human dental pulp cells (hDPCs)
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Dentistry
Link
https://link.springer.com/content/pdf/10.1007/s00784-023-05466-5.pdf
Reference79 articles.
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2. Duncan HF, Smith AJ, Fleming GJP, Cooper PR (2012) Histone deacetylase inhibitors induced differentiation and accelerated mineralization of pulp-derived cells. J Endod 38(3):339–345. https://doi.org/10.1016/j.joen.2011.12.014
3. Kearney M, Cooper PR, Smith AJ, Duncan HF (2018) Epigenetic approaches to the treatment of dental pulp inflammation and repair: opportunities and obstacles. Front Genet 9:1–18. https://doi.org/10.3389/fgene.2018.00311
4. Paino F, La Noce A, Tirino V, Naddeo A, Desiderio V, Pirozzi U et al (2014) Histone deacetylase inhibition with valproic acid downregulates osteocalcin gene expression in human dental pulp stem cells and osteoblasts: Evidence for HDAC2 involvement. Stem Cells 32(1):279–289. https://doi.org/10.1002/stem.1544
5. Jin H, Park JY, Choi H, Choung PH (2013) HDAC inhibitor trichostatin a promotes proliferation and odontoblast differentiation of human dental pulp stem cells. Tissue Eng - Part A 19(5–6):613–624. https://doi.org/10.1089/ten.TEA.2012.0163
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