A Transient Decrease in Spleen Size Following Stroke Corresponds to Splenocyte Release into Systemic Circulation-Reference-Cited by-同舟云学术

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A Transient Decrease in Spleen Size Following Stroke Corresponds to Splenocyte Release into Systemic Circulation

Published:2012-10-06 Issue:4 Volume:7 Page:1017-1024
ISSN:1557-1890
Container-title:Journal of Neuroimmune Pharmacology
language:en
Short-container-title:J Neuroimmune Pharmacol

Author:

Seifert Hilary A.,Hall Aaron A.,Chapman Cortney B.,Collier Lisa A.,Willing Alison E.,Pennypacker Keith R.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology,Immunology and Allergy,Neuroscience (miscellaneous)

Link

http://link.springer.com/content/pdf/10.1007/s11481-012-9406-8.pdf

Reference37 articles.

1. Ajmo CT Jr, Vernon DO, Collier L, Pennypacker KR, Cuevas J (2006) Sigma receptor activation reduces infarct size at 24 hours after permanent middle cerebral artery occlusion in rats. Curr Neurovasc Res 3(2):89–98

2. Ajmo CT Jr, Vernon DO, Collier L, Hall AA, Garbuzova-Davis S, Willing A, Pennypacker KR (2008) The spleen contributes to stroke-induced neurodegeneration. J Neurosci Res 86:2227–2234

3. Ajmo CT Jr., Collier LA, Leonardo CC, Hall AA, Green SM, Womble TA, Cuevas J, Willing AE, Pennypacker KR (2009) Blockade of adrenoreceptors inhibits the splenic response to stroke. Exp Neurol

4. Bakovic D, Eterovic D, Saratlija-Novakovic Z, Palada I, Valic Z, Bilopavlovic N, Dujic Z (2005) Effect of human splenic contraction on variation in circulating blood cell counts. Clin Exp Pharmacol Physiol 32(11):944–951

5. Bao Y, Kim E, Bhosle S, Mehta H, Cho S (2010) A role for spleen monocytes in post-ischemic brain inflammation and injury. J Neuroinflammation 7:92

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