Inhibition of the Dead Box RNA Helicase 3 Prevents HIV-1 Tat and Cocaine-Induced Neurotoxicity by Targeting Microglia Activation
Author:
Funder
NIH NIDA
NIH NCI
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology,Immunology and Allergy,Neuroscience (miscellaneous)
Link
http://link.springer.com/content/pdf/10.1007/s11481-019-09885-8.pdf
Reference100 articles.
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2. Aksenov MY, Aksenova MV, Nath A, Ray PD, Mactutus CF, Booze RM (2006) Cocaine-mediated enhancement of Tat toxicity in rat hippocampal cell cultures: the role of oxidative stress and D1 dopamine receptor. Neurotoxicology 27:217–228. https://doi.org/10.1016/j.neuro.2005.10.003
3. Aksenova MV, Aksenov MY, Adams SM, Mactutus CF, Booze RM (2009) Neuronal survival and resistance to HIV-1 Tat toxicity in the primary culture of rat fetal neurons. Exp Neurol 215:253–263. https://doi.org/10.1016/j.expneurol.2008.10.006
4. Al-Harti L, Joseph J, Nath A (2018) Astrocytes as an HIV CNS reservoir: highlights and reflections of an NIMH-sponsored symposium. J Neurovirol 24:665–669. https://doi.org/10.1007/s13365-018-0691-8
5. Ariumi Y (2014) Multiple functions of DDX3 RNA helicase in gene regulation, tumorigenesis, and viral infection. Front Genet 5:423. https://doi.org/10.3389/fgene.2014.00423
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