Role of complement in the pathogenesis of thrombotic microangiopathies
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Hematology
Link
http://link.springer.com/article/10.1007/s12254-017-0380-y/fulltext.html
Reference31 articles.
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2. Szarvas N, et al. Genetic analysis and functional characterization of novel mutations in a series of patients with atypical hemolytic uremic syndrome. Mol Immunol. 2016;71:10–22.
3. Caprioli J, et al. Genetics of HUS: the impact of MCP, CFH, and IF mutations on clinical presentation, response to treatment, and outcome. Blood. 2006;108(4):1267–79.
4. Westra D, et al. Atypical hemolytic uremic syndrome and genetic aberrations in the complement factor H‑related 5 gene. J Hum Genet. 2012;57(7):459–64.
5. Jozsi M, et al. Factor H autoantibodies in atypical hemolytic uremic syndrome correlate with CFHR1/CFHR3 deficiency. Blood. 2008;111(3):1512–4.
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1. Elevated Systemic Pentraxin-3 Is Associated With Complement Consumption in the Acute Phase of Thrombotic Microangiopathies;Frontiers in Immunology;2019-02-25
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