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Alterations and abnormal expression of A20 in peripheral monocyte subtypes in patients with rheumatoid arthritis

  • Published:2020-06-02 Issue:1 Volume:40 Page:341-348
  • ISSN:0770-3198
  • Container-title:Clinical Rheumatology
  • language:en
  • Short-container-title:Clin Rheumatol

Author:

Zhang LuORCID,Yao Yao,Tian Junmei,Jiang Wanlan,Zhou Shiliang,Chen Jinyun,Xu Ting,Wu Min

Funder

the National Natural Science Foundation of China

Scientific Research Projects for Top-notch Talents in Jiangsu Province

Jiangsu Postdoctoral Research Foundation

Graduate Research and Innovation Projects of Jiangsu Province

Science and Technology Project of Changzhou Health Committee for Young Talents

the Key Programs of Changzhou City Commission for Discipline Inspection

the Research Project of Jiangsu Modern Hospital Management

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Rheumatology

Reference29 articles.

1. Ciccacci C, Latini A, Perricone C, Conigliaro P, Colafrancesco S, Ceccarelli F, Priori R, Conti F, Perricone R, Novelli G, Borgiani P (2019) TNFAIP3 gene polymorphisms in three common autoimmune diseases: systemic lupus erythematosus, rheumatoid arthritis, and primary Sjogren syndrome-association with disease susceptibility and clinical phenotypes in Italian patients. J Immunol Res 2019:6728694–6728696. https://doi.org/10.1155/2019/6728694

2. López-Zambrano M, Rodriguez-Montesinos J, Crespo-Avilan GE, Muñoz-Vega M, Preissner KT (2020) Thrombin promotes macrophage polarization into M1-like phenotype to induce inflammatory responses. Thromb Haemost 120:658–670. https://doi.org/10.1055/s-0040-1703007

3. Zhao C, Zhang L, Kong W, Liang J, Xu X, Wu H, Feng X, Hua B, Wang H, Sun L (2015) Umbilical cord-derived mesenchymal stem cells inhibit cadherin-11 expression by fibroblast-like synoviocytes in rheumatoid arthritis. J Immunol Res 2015:137695–137610. https://doi.org/10.1155/2015/137695

4. Kapellos TS, Bonaguro L, Gemünd I, Reusch N, Saglam A, Hinkley ER, Schultze JL (2019) Human monocyte subsets and phenotypes in major chronic inflammatory diseases. Front Immunol 10:2035. https://doi.org/10.3389/fimmu.2019.02035

5. Lacerte P, Brunet A, Egarnes B, Duchêne B, Brown JP, Gosselin J (2016) Overexpression of TLR2 and TLR9 on monocyte subsets of active rheumatoid arthritis patients contributes to enhance responsiveness to TLR agonists. Arthritis Res Ther 18:10. https://doi.org/10.1186/s13075-015-0901-1
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