IRAK2 is associated with susceptibility to rheumatoid arthritis-Reference-Cited by-同舟云学术

IRAK2 is associated with susceptibility to rheumatoid arthritis

Published:2017-11-11 Issue:4 Volume:37 Page:927-933
ISSN:0770-3198
Container-title:Clinical Rheumatology
language:en
Short-container-title:Clin Rheumatol

Author:

Hassine Hana Ben,Sghiri Rim,Chabchoub Elyes,Boumiza Asma,Slama Foued,Baccouche Khadija,Shakoor Zahid,Almogren Adel,Mariaselvam Christina,Tamouza Ryad,Bouajina Elyes,Zemni Ramzi

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Rheumatology

Link

http://link.springer.com/article/10.1007/s10067-017-3906-0/fulltext.html

Reference37 articles.

1. Sun SC, Chang JH, Jin J (2013) Regulation of NF-kB in autoimmunity. Trends Immunol 34:282–289

2. Cho ML, Kang JW, Moon YM et al (2006) STAT3 and NF-kB signal pathway is required for IL-23-mediated IL-17 production in spontaneous arthritis animal model IL-1receptor antagonist- deficient mice. J Immunol 176:5652–5661

3. Yang XO, Pappu BP, Nurieva R et al (2008) T helper17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma. Immunity 28:29–39

4. Bondeson J, Foxwell B, Brennan F et al (1999) Defining therapeutic targets by using adenovirus: blocking NF-kB inhibits both inflammatory and destructive mechanisms in rheumatoid synovium but spares anti-inflammatory mediators. Proc Natl Acad Sci U S A 96:5668–5673

5. Miagkov AV, Kovalenko DV, Brown CE et al (1998) NF-kB activation provides the potential link between inflammation and hyperplasia in the arthritic joint. Proc Natl Acad Sci U S A 95:13859–13864

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