Senescent CD4+CD28null cells are increased in chronic hyperuricemia, show aberrant effector phenotypes, and are reversed after allopurinol therapy: a proof-of-concept pilot study
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Rheumatology
Link
https://link.springer.com/content/pdf/10.1007/s10067-023-06595-8.pdf
Reference22 articles.
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2. Zizzo G, De Santis M, Bosello S, Tolusso B, Alivernini S, De Luca G, Gremese E, Ferraccioli G (2019) Measuring the T-cell down-regulation of TCR-zeta, ZAP-70 and CD28 in arthritis patients: an old tool for new biomarkers. Eur J Immunol 49:2195–2203. https://doi.org/10.1002/eji.201847849
3. Dumitriu IE (2015) The life (and death) of CD4+ CD28(null) T cells in inflammatory diseases. Immunology 146:185–193. https://doi.org/10.1111/imm.12506
4. Lozada-Navarro AC, Castillo-Martínez D, Moreno-Ramírez M, Acosta-Peña G, Páez A, Massó F, Márquez-Velasco R, Amezcua-Guerra LM (2018) An imbalance in the T-helper phenotypes displayed by senescent CD4+CD28null T cells is associated with erosive arthritis (rhupus syndrome) in systemic lupus erythematosus. Lupus 27:2155–2160. https://doi.org/10.1177/0961203318793715
5. Gómez-García L, Ramírez-Assad C, Vargas A, Massó F, Sánchez-Muñoz F, Márquez-Velasco R, Amezcua-Guerra LM, Bojalil R (2013) Reduced numbers of circulating CD28-negative CD4+ cells in patients with rheumatoid arthritis chronically treated with abatacept. Int J Rheum Dis 16:469–471. https://doi.org/10.1111/1756-185X.12056
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