Genetischer Polymorphismus in der G-Protein-β3-Untereinheit, Adipositas und essentielle Hypertonie
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
http://link.springer.com/content/pdf/10.1007/BF03044121.pdf
Reference60 articles.
1. 1999 World Health Organization-International Society of Hypertension Guidelines for the Management of Hypertension. Guidelines Subcommittee. J Hypertens 1999;17:151–83.
2. Baumgart D, Naber C, Haude M, et al. G protein β3 subunit 825T-allele and enhanced coronary vasoconstriction on α2-adrenoceptor activation. Circ Res 1999;85:965–9.
3. Beige J, Hohenbleicher H, Distler A, et al. G-protein beta3 subunit C825T variant and ambulatory blood pressure in essential hypertension. Hypertension 1999;33:1049–51.
4. Benjafield AV, Jeyasingam CL, Nyholt DR, et al. G-Protein beta3 subunit gene (GNB3) variant in causation of essential hypertension. Hypertension 1998;32:1094–7.
5. Bourne HR. How receptors talk to trimeric G proteins. Curr Opin Cell Biol 1997;9:134–42.
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1. Association between GNB3 c.825C > T polymorphism and the risk of overweight and obesity: A meta-analysis;Meta Gene;2016-09
2. Ablation of the GNB3 gene in mice does not affect body weight, metabolism or blood pressure, but causes bradycardia;Cellular Signalling;2014-11
3. Association of the GNB3 825T-allele with better survival in patients with glioblastoma multiforme;Journal of Cancer Research and Clinical Oncology;2010-02-10
4. G protein polymorphisms do not predict weight loss and improvement of hypertension in severely obese patients;Journal of Gastrointestinal Surgery;2004-11-01
5. SSRIs antidepressant activity is influenced by Gβ3 variants;European Neuropsychopharmacology;2003-03
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