Angiogenin as a Possible Mediator of Macrophage-Mediated Regulation of Fibroblast Functions
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10517-023-05921-z.pdf
Reference14 articles.
1. Witherel CE, Abebayehu D, Barker TH, Spiller KL. Macrophage and fibroblast interactions in biomaterial-mediated fibrosis. Adv. Healthc Mater. 2019;8(4):e1801451. doi: https://doi.org/10.1002/adhm.201801451
2. Pakshir P, Hinz B. The big five in fibrosis: Macrophages, myofibroblasts, matrix, mechanics, and miscommunication. Matrix Biol. 2018;68-69:81-93. doi: https://doi.org/10.1016/j.matbio.2018.01.019
3. Poto R, Loffredo S, Palestra F, Marone G, Patella V, Varricchi G. Angiogenesis, lymphangiogenesis, and inflammation in Chronic Obstructive Pulmonary Disease (COPD): few certainties and many outstanding questions. Cells. 2022;11(10):1720. doi: https://doi.org/10.3390/cells11101720
4. Sheng J, Xu Z. Three decades of research on angiogenin: a review and perspective. Acta Biochim. Biophys. Sin. (Shanghai). 2016;48(5):399-410. doi: https://doi.org/10.1093/abbs/gmv131
5. Lee SH, Kim KW, Min KM, Kim KW, Chang SI, Kim JC. Angiogenin reduces immune inflammation via inhibition of TANK-binding kinase 1 expression in human corneal fibroblast cells. Mediators Inflamm. 2014;2014:861435. doi: https://doi.org/10.1155/2014/861435
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