The Role of Mitochondrial KATP Channels in the Infarct-Reducing Effect of Normobaric Hypoxia
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10517-023-05671-y.pdf
Reference9 articles.
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2. Koppen E, Madsen E, Greiff G, Stenseth R, Pleym H, Wiseth R, Wahba A, Videm V. Perioperative factors associated with changes in Troponin T during coronary artery bypass grafting. J. Cardiothorac. Vasc. Anesth. 2019;33(12):3309-3319. https://doi.org/10.1053/j.jvca.2019.06.029
3. Heusch G. Molecular basis of cardioprotection: signal transduction in ischemic pre-, post-, and remote conditioning. Circ. Res. 2015;116(4):674-699. https://doi.org/10.1161/CIRCRESAHA.116.305348
4. Yellon DM, Downey JM. Preconditioning the myocardium: from cellular physiology to clinical cardiology. Physiol. Rev. 2003;83(4):1113-1151. https://doi.org/10.1152/physrev.00009.2003
5. Maslov LN, Naryzhnaia NV, Tsibulnikov SY, Kolar F, Zhang Y, Wang H, Gusakova AM, Lishmanov YB. Role of endogenous opioid peptides in the infarct size-limiting effect of adaptation to chronic continuous hypoxia. Life Sci. 2013;93(9-11):373-379. https://doi.org/10.1016/j.lfs.2013.07.018
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