Na+ leak-current channel (NALCN) at the junction of motor and neuropsychiatric symptoms in Parkinson’s disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Biological Psychiatry,Psychiatry and Mental health,Clinical Neurology,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s00702-021-02348-6.pdf
Reference153 articles.
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3. Adayey T, Chen-Hwang MC, Murakami N, Wang R, Hwang YW (2006) MNB/DYRK1A phosphorylation regulates the interactions of synaptojanin 1 with endocytic accessory proteins. Biochem Biophys Res Commun 351:1060–1065
4. Al-Sayed MD, Al-Zaiden H, Albakheet A et al (2013) Mutations in NALCN cause an autosomal-recessive syndrome with severe hypotonia, speech impairment, and cognitive delay. Am J Hum Genet 93:721–726
5. Anderson JP, Walker DE, Goldstein JM et al (2006) Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease. J Biol Chem 281:29739–29752
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