Rasagiline and selegiline suppress calcium efflux from mitochondria by PK11195-induced opening of mitochondrial permeability transition pore: a novel anti-apoptotic function for neuroprotection-Reference-Cited by-同舟云学术

Rasagiline and selegiline suppress calcium efflux from mitochondria by PK11195-induced opening of mitochondrial permeability transition pore: a novel anti-apoptotic function for neuroprotection

Published:2015-04-12 Issue:10 Volume:122 Page:1399-1407
ISSN:0300-9564
Container-title:Journal of Neural Transmission
language:en
Short-container-title:J Neural Transm

Author:

Wu Yuqiu,Kazumura Kimiko,Maruyama Wakako,Osawa Toshihiko,Naoi Makoto

Publisher

Springer Science and Business Media LLC

Subject

Biological Psychiatry,Psychiatry and Mental health,Neurology (clinical),Neurology

Link

http://link.springer.com/content/pdf/10.1007/s00702-015-1398-0.pdf

Reference56 articles.

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2. Azarashvili T, Grachev G, Krestinina O, Evtodienko Y, Yurkov I et al (2007) The peripheral-type benzodiazepine receptor is involved in control of Ca2+-induced permeability transition pore opening in rat brain mitochondria. Cell Calcium 42:27–39

3. Baumgartner HK, Gerasimenko JV, Thorne C, Ferdek P, Pozzan T et al (2009) Calcium elevation in mitochondria is requirement for mitochondrial permeability transition pore (mPTP) opening. J Biol Chem 284:20796–20803

4. Bernardi P (1996) The permeability transition pore. Control of a cyclosporine A-sensitive mitochondrial channel involved in cell death. Biochim Biophys Acta 1275:5–9

5. Bernardi P, von Stockum S (2012) The permeability transition pore as a Ca2+ channel: new answers to an old question. Cell Calcium 52:22–27

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