Role of Vasoactive Intestinal Peptide in Hyperoxia-Induced Injury of Primary Type II Alveolar Epithelial Cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology and Child Health
Link
http://link.springer.com/content/pdf/10.1007/s12098-010-0248-1.pdf
Reference18 articles.
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2. Pagano A, Barazzone-Argiroffo C. Alveolar cell death in hyperoxia-Induced lung injury. Ann NY Acad Sci. 2003;1010:405–16.
3. Miyake Y, Kaise H, Isono K, Koseki H, Kohno K, Tanaka M. Protective role of macrophages in noninflammatory lung injury caused by selective ablation of alveolar epithelial type II cells. J Immunol. 2007;178:5001–9.
4. Mantell LL, Lee PJ. Signal transduction pathway in hyperoxia-induced lung cell death. Mol Genet Metab. 2000;71:359–70.
5. Onoue S, Yamada S, Yajima T. Bioactive analogues and drug delivery systems of vasoactive intestinal peptide (VIP) for the treatment of asthma/COPD. Peptides. 2007;28:1640–50.
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3. CGRP attenuates hyperoxia-induced oxidative stress-related injury to alveolar epithelial type II cells via the activation of the Sonic hedgehog pathway;International Journal of Molecular Medicine;2017-05-26
4. Prevention of hyperoxia-induced bronchial hyperreactivity by sildenafil and vasoactive intestinal peptide: impact of preserved lung function and structure;Respiratory Research;2014-08-13
5. Vasoactive intestinal peptide reduces oxidative stress in pancreatic acinar cells through the inhibition of NADPH oxidase;Peptides;2011-10
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