High expression of a novel splicing variant of VEGF, L-VEGF144 in glioblastoma multiforme is associated with a poorer prognosis in bevacizumab treatment-Reference-Cited by-同舟云学术

High expression of a novel splicing variant of VEGF, L-VEGF144 in glioblastoma multiforme is associated with a poorer prognosis in bevacizumab treatment

Published:2018-06-16 Issue:1 Volume:140 Page:37-47
ISSN:0167-594X
Container-title:Journal of Neuro-Oncology
language:en
Short-container-title:J Neurooncol

Author:

Cheng Wen-Yu^ORCID,Shen Chiung-Chyi,Chiao Ming-Tsang,Liang Yea-Jiuan,Mao Tsuo-Fei,Liu Bai-Shuan,Chen Jun-Peng

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Clinical Neurology,Neurology,Oncology

Link

http://link.springer.com/article/10.1007/s11060-018-2928-z/fulltext.html

Reference17 articles.

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2. Lu KV, Bergers G (2013) Mechanisms of evasive resistance to anti-VEGF therapy in glioblastoma. CNS Oncol 2:49–65. https://doi.org/10.2217/cns.12.36

3. Shen CC, Cheng WY, Chiao MT, Liang YJ, Mao TF, Liu BS (2016) Two novel heparin-binding vascular endothelial growth factor splices, L-VEGF144 and L-VEGF138, are expressed in human glioblastoma cells. Curr Neurovasc Res 13:207–218

4. Zhang HT, Scott PA, Morbidelli L, Peak S, Moore J, Turley H, Harris AL, Ziche M, Bicknell R (2000) The 121 amino acid isoform of vascular endothelial growth factor is more strongly tumorigenic than other splice variants in vivo. Br J Cancer 83:63–68. https://doi.org/10.1054/bjoc.2000.1279

5. Huez I, Bornes S, Bresson D, Creancier L, Prats H (2001) New vascular endothelial growth factor isoform generated by internal ribosome entry site-driven CUG translation initiation. Mol Endocrinol 15:2197–2210. https://doi.org/10.1210/mend.15.12.0738

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