Identification of potential key lipid metabolism-related genes involved in tubular injury in diabetic kidney disease by bioinformatics analysis
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s00592-024-02278-1.pdf
Reference42 articles.
1. Alicic RZ, Rooney MT, Tuttle KR (2017) Diabetic kidney disease: challenges, progress, and possibilities. Clin J Am Soc Nephrol 12(12):2032–2045
2. Yang W, Luo Y, Yang S et al (2018) Ectopic lipid accumulation: potential role in tubular injury and inflammation in diabetic kidney disease. Clin Sci (Lond) 132(22):2407–2422
3. Kimmelstiel P, Wilson C (1936) Intercapillary lesions in the glomeruli of the Kidney. Am J Pathol 12(1):83–98
4. Bai Y, Ma L, Deng D et al (2023) Bioinformatic identification of genes involved in diabetic nephropathy fibrosis and their clinical relevance. Biochem Genet 61(4):1567–1584
5. Mai L, He G, Chen J et al (2023) Profilin1 promotes renal tubular epithelial cell apoptosis in diabetic nephropathy through the Hedgehog signaling pathway. Diabetes Metab Syndr Obes 16:1731–1743
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