Azelnidipine protects HL-1 cardiomyocytes from hypoxia/reoxygenation injury by enhancement of NO production independently of effects on gene expression-Reference-Cited by-同舟云学术

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Azelnidipine protects HL-1 cardiomyocytes from hypoxia/reoxygenation injury by enhancement of NO production independently of effects on gene expression

Published:2024-05-26 Issue: Volume: Page:
ISSN:0910-8327
Container-title:Heart and Vessels
language:en
Short-container-title:Heart Vessels

Author:

Minato Hiroyuki,Endo Ryo,Kurata Yasutaka,Notsu Tomomi,Kinugasa Yoshiharu,Wakimizu Takayuki,Tsuneto Motokazu,Shirayoshi Yasuaki,Ninomiya Haruaki,Yamamoto Kazuhiro,Hisatome Ichiro,Otsuki Akihiro

Funder

Japan Society for the Promotion of Science

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s00380-024-02415-4.pdf

Reference30 articles.

1. Kunst G, Klein AA (2015) Peri-operative anaesthetic myocardial preconditioning and protection—cellular mechanisms and clinical relevance in cardiac anaesthesia. Anaesthesia 70:467–482

2. Tsai SK, Lin SM, Huang CH, Hung WC, Chih CL, Huang SS (2004) Effect of desflurane-induced preconditioning following ischemia-reperfusion on nitric oxide release in rabbits. Life Sci 76:651–660

3. Schulz R, Kelm M, Heusch G (2004) Nitric oxide in myocardial ischemia/reperfusion injury. Cardiovasc Res 61:402–413

4. Cohen MV, Yang XM, Downey JM (2006) Nitric oxide is a preconditioning mimetic and cardioprotectant and is the basis of many available infarct-sparing strategies. Cardiovasc Res 70:231–239

5. Bice JS, Jones BR, Chamberlain GR, Baxter GF (2016) Nitric oxide treatments as adjuncts to reperfusion in acute myocardial infarction: a systematic review of experimental and clinical studies. Basic Res Cardiol 111:23

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