Lack of association of TLR4 gene Asp299Gly and Thr399Ile polymorphisms with rheumatoid arthritis in Chinese Han population of Yunnan Province-Reference-Cited by-同舟云学术

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Lack of association of TLR4 gene Asp299Gly and Thr399Ile polymorphisms with rheumatoid arthritis in Chinese Han population of Yunnan Province

Published:2010-03-20 Issue:9 Volume:30 Page:1249-1252
ISSN:0172-8172
Container-title:Rheumatology International
language:en
Short-container-title:Rheumatol Int

Author:

Zheng Bingrong,Li Qin,Wei Chuanyu,Qin Jiao,Shou Tao,Zhou Ruoyu,Shao Jingru,Yang Yan,Xiao Chunjie

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy,Rheumatology

Link

http://link.springer.com/content/pdf/10.1007/s00296-010-1400-y.pdf

Reference33 articles.

1. Lawrence JS, Heberden Oration (1970) 1969: rheumatoid arthritis: nature or nurture? Ann Rheum Dis 29:357–379

2. Orozco G, Rueda B, Martin J (2006) Genetic basis of rheumatoid arthritis. Biomed Pharmacother 60:656–662

3. Wordsworth BP, Lanchbury JS, Sakkas LI, Welsh KI, Panayi GS, Bell JI (1989) HLA-DR4 subtype frequencies in rheumatoid arthritis indicate that DRB1 is the major susceptibility locus within the HLA classII region. Proc Natl Acad Sci 86:10049–10053

4. Deighton CM, Walker DJ, Griffiths ID, Roberts DF (1989) The contribution of HLA to rheumatoid arthritis. Clin Genet 36:178–182

5. Radstake TR, Franke B, Hanssen S, Netea MG, Welsing P, Barrera P et al (2004) The toll-like receptor 4 Asp299Gly functional variant is associated with decreased rheumatoid arthritis disease susceptibility but does not influence disease severity and/or outcome. Arthritis Rheum 50:999–1001

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