Glucose transporters and glucose utilization in rat brain after acute ethanol administration-Reference-Cited by-同舟云学术

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Glucose transporters and glucose utilization in rat brain after acute ethanol administration

Published:2000-09 Issue:3 Volume:15 Page:211-222
ISSN:0885-7490
Container-title:Metabolic Brain Disease
language:en
Short-container-title:Metab Brain Dis

Author:

Handa Raj K.,DeJoseph Mary R.,Singh Leela D.,Hawkins Richard A.,Singh Sant P.

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/BF02674530.pdf

Reference44 articles.

1. Auer, R. N., and Anderson, L. G. (1996). Hypoglycemic brain damage: effect of a dihydropyridine calcium channel antagonist in rats.Diabetologia 39:129–134.

2. Avruch, J., and Wallach, D. F. (1971). Preparation and properties of plasma membrane and endoplasmic reticulum fragments from isolated rat fat cells.Biochim. Biophys. Acta 233:334–347.

3. Brndsted, H. E., Reith, J., and Gjedde, A. (1990). Regional glucose phosphorylation rate in rat brain during acute ethanol intoxication.Acta Neurol. Scand. 81:229–232.

4. Clemmesen, L., Jorgensen, O. S., Hemmingsen, R., Barry, D. I., and Bolwig, T. G. (1987). Brain marker protein changes after short and long-term ethanol intoxication and withdrawal in the rat.J. Psychiatr. Res. 21:171–183.

5. De Feo, P. (1988). Modest decrements in plasma glucose concentration cause early impairment in cognitive function and later activation of glucose counterregulation in the absence of hypoglycemic symptoms in normal man.J. Clin. Invest. 82:436–444.

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