The role of nitric oxide in nociception
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11916-000-0070-y.pdf
Reference50 articles.
1. Geller DA, Billiar TR: Molecular biology of nitric oxide synthases. Cancer Metastasis Rev 1998, 17:7–23. Provides a thorough review on the latest advances in our understanding of molecular regulation of the NOS genes.
2. Wang Y, Marsden PA: Nitric oxide synthases: gene structure and regulation. Adv Pharmacol 1995, 34:71–90.
3. Lewin MR, Walters ET: Cyclic GMP pathway is critical for inducing long-term sensitization of nociceptive sensory neurons. Nat Neurosci 1999, 2:18–23. Using electrophysiologic and pharmacologic approches, this study demonstrates that the NO-cGMP-protein kinase pathway is required for noxious stimulation-induced long-term hyperexcitability of nociceptive sensory neurons in the mollusc Aplysia.
4. Lin Q, Palecek J, Paleckova V, et al.: Nitric oxide mediates the central sensitization of primate spinothalamic tract neurons. J Neurophysiol 1999, 81:1075–85. Provides in vivo evidence to link spinal NO production to central sensitization and tissue injury-related pain states.
5. Lin Q, Wu J, Peng YB, et al.: Nitric oxide-mediated spinal disinhibition contributes to the sensitization of primate spinothalamic tract neurons. J Neurophysiol 1999, 81:1086–1094. Suggests that a NO-mediated pathway may contribute to central sensitization through a mechanism of disinhibition.
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