Animal models for researching approaches to therapy of Duchenne muscular dystrophy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Agronomy and Crop Science,Genetics,Animal Science and Zoology,Biotechnology
Link
https://link.springer.com/content/pdf/10.1007/s11248-021-00278-3.pdf
Reference135 articles.
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2. Allen DG, Whitehead NP (2010) Duchenne muscular dystrophy–what causes the increased membrane permeability in skeletal muscle? Int J Biochem Cell Biol 43:290–294. https://doi.org/10.1016/j.biocel.2010.11.005
3. Allen DG, Whitehead NP, Froehner SC (2016) Absence of dystrophin disrupts skeletal muscle signaling: roles of Ca2+, reactive oxygen species, and nitric oxide in the development of muscular dystrophy. Physiol Rev 96:253–305. https://doi.org/10.1152/physrev.00007.2015
4. Ambrósio CE, Valadares MC, Zucconi E, Cabral R, Pearson PL, Gaiad TP, Canovas M, Vainzof M, Miglino MA, Zatz M (2008) Ringo, a golden retriever muscular dystrophy (GRMD) dog with absent dystrophin but normal strength. Neuromuscul Disord 18:892–893. https://doi.org/10.1016/j.nmd.2008.06.385
5. Amoasii L, Long C, Li H, Mireault AA, Shelton JM, Sanchez-Ortiz E, McAnally JR, Bhattacharyya S, Schmidt F, Grimm D, Hauschka SD, Bassel-Duby R, Olson EN (2017) Single-cut genome editing restores dystrophin expression in a new mouse model of muscular dystrophy. Sci Transl Med 9:eaan081. https://doi.org/10.1126/scitranslmed.aan8081
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