Quercetin and dasatinib, two powerful senolytics in age-related cardiovascular disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Geriatrics and Gerontology,Gerontology,Aging
Link
https://link.springer.com/content/pdf/10.1007/s10522-023-10068-5.pdf
Reference73 articles.
1. Alejandro S-P (2022) ER stress in cardiac aging, a current view on the d-galactose model. Exp Gerontol. 169:111953–55. https://doi.org/10.1016/j.exger.2022.111953
2. Anderson R, Lagnado A, Maggiorani D et al (2019) Length-independent telomere damage drives post-mitotic cardiomyocyte senescence. EMBO J. https://doi.org/10.15252/embj.2018100492
3. Angeliki V, Warren JS, Drosatos K (2023) Cardiovascular aging: from cellular and molecular changes to therapeutic interventions. J Cardiovasc Aging 3:23. https://doi.org/10.20517/jca.2023.09
4. Baggett BC, Murphy KR, Sengun E et al (2023) Myofibroblast senescence promotes arrhythmogenic remodeling in the aged infarcted rabbit heart. Elife. https://doi.org/10.7554/eLife.84088
5. Baker DJ, Wijshake T, Tchkonia T et al (2011) Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders. Nature 479:232–236. https://doi.org/10.1038/nature10600
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