The post translational modification of key regulators of ATR signaling in DNA replication
Author:
Funder
National Natural Science Foundation
Shanghai Pujiang program
China Postdoctoral Science Foundation
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s42764-021-00036-z.pdf
Reference125 articles.
1. Alonso-de Vega, I., Martín, Y., & Smits, V. A. (2014). USP7 controls Chk1 protein stability by direct deubiquitination. Cell Cycle (Georgetown, Tex.), 13, 3921–3926. https://doi.org/10.4161/15384101.2014.973324
2. Anantha, R. W., Vassin, V. M., & Borowiec, J. A. (2007). Sequential and synergistic modification of human RPA stimulates chromosomal DNA repair. The Journal of Biological Chemistry, 282, 35910–35923. https://doi.org/10.1074/jbc.M704645200
3. Ardito, F., Giuliani, M., Perrone, D., Troiano, G., & Lo Muzio, L. (2017). The crucial role of protein phosphorylation in cell signaling and its use as targeted therapy (Review). International Journal of Molecular Medicine, 40, 271–280. https://doi.org/10.3892/ijmm.2017.3036
4. Ashley, A. K., et al. (2014). DNA-PK phosphorylation of RPA32 Ser4/Ser8 regulates replication stress checkpoint activation, fork restart, homologous recombination and mitotic catastrophe. DNA Repair, 21, 131–139. https://doi.org/10.1016/j.dnarep.2014.04.008
5. Awasthi, P., Foiani, M., & Kumar, A. (2015). ATM and ATR signaling at a glance. Journal of Cell Science, 128, 4255–4262. https://doi.org/10.1242/jcs.169730
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