Exploring the DNA damage response pathway for synthetic lethality-Reference-Cited by-同舟云学术

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Exploring the DNA damage response pathway for synthetic lethality

Published:2022-11-02 Issue:2 Volume:4 Page:98-120
ISSN:2524-7662
Container-title:Genome Instability & Disease
language:en
Short-container-title:GENOME INSTAB. DIS.

Author:

Xu Xin,Nowsheen Somaira,Deng Min

Funder

Central Research Institute Fund of Chinese Academy of Medical Sciences

Beijing Hope Run Special Fund of Cancer Foundation of China

CAMS Innovation Fund for Medical Sciences

Publisher

Springer Science and Business Media LLC

Subject

General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s42764-022-00087-w.pdf

Reference192 articles.

1. Adames, N. R., Gallegos, J. E., & Peccoud, J. (2019). Yeast genetic interaction screens in the age of CRISPR/Cas. Current Genetics, 65(2), 307–327.

2. Ahlskog, J. K., Larsen, B. D., Achanta, K., & Sørensen, C. S. (2016). ATM/ATR-mediated phosphorylation of PALB2 promotes RAD51 function. EMBO Reports, 17(5), 671–681.

3. Álvarez-Quilón, A., et al. (2020). Endogenous DNA 3’ Blocks Are Vulnerabilities for BRCA1 and BRCA2 deficiency and are reversed by the APE2 nuclease. Molecular Cell, 78(6), 1152-1165.e1158.

4. Anastasia, A., et al. (2022). The DNA-PK Inhibitor AZD7648 Sensitizes patient-derived ovarian cancer xenografts to pegylated liposomal doxorubicin and olaparib preventing abdominal metastases. Molecular Cancer Therapeutics, 21(4), 555–567.

5. Audeh, M. W., et al. (2010). Oral poly(ADP-ribose) polymerase inhibitor olaparib in patients with BRCA1 or BRCA2 mutations and recurrent ovarian cancer: a proof-of-concept trial. Lancet (london, England), 376(9737), 245–251.

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