Reply to the letter titled “More likely than through head trauma, is LHON triggered by mitochondrion-toxic drugs or oxidative stress”
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Sensory Systems,Ophthalmology
Link
https://link.springer.com/content/pdf/10.1007/s10633-020-09816-6.pdf
Reference6 articles.
1. Kim HD (2020) Leber hereditary optic neuropathy following head trauma and ocular trauma on contralateral eye: a case report. Doc Ophthalmol. https://doi.org/10.1007/s10633-020-09801-z
2. Giordano L, Deceglie S, d’Adamo P et al (2015) Cigarette toxicity triggers Leber’s hereditary optic neuropathy by affecting mtDNA copy number, oxidative phosphorylation and ROS detoxification pathways. Cell Death Dis 6:e2021. https://doi.org/10.1038/cddis.2015.364
3. Yu-Wai-Man P, Griffiths PG, Chinnery PF (2011) Mitochondrial optic neuropathies—disease mechanisms and therapeutic strategies. Prog Retin Eye Res 30:81–114. https://doi.org/10.1016/j.preteyeres.2010.11.002
4. Kirkman MA, Yu-Wai-Man P, Korsten A et al (2009) Gene-environment interactions in Leber hereditary optic neuropathy. Brain 132(9):2317–2326. https://doi.org/10.1093/brain/awp158
5. Nagai A, Nakamura M, Kusuhara S, Kanamori A, Negi A (2005) Unilateral manifestation of Leber’s hereditary optic neuropathy after blunt ocular trauma. Jpn J Ophthalmol 49:65–67. https://doi.org/10.1007/s10384-004-0140-5
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