Fundus autofluorescence, optical coherence tomography and electroretinography abnormalities in a patient with digoxin retinopathy that resemble those in KCNV2-associated retinopathy
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Sensory Systems,Ophthalmology
Link
https://link.springer.com/content/pdf/10.1007/s10633-023-09942-x.pdf
Reference11 articles.
1. Madreperla SA, Johnson MA, Nakatani K (1994) Electrophysiologic and electroretinographic evidence for photoreceptor dysfunction as a toxic effect of digoxin. Arch Ophthalmol 112:807–812. https://doi.org/10.1001/archopht.1994.01090180105044
2. Kinoshita J, Iwata N, Kimotsuki T, Yasuda M (2014) Digoxin-induced reversible dysfunction of the cone photoreceptors in monkeys. Invest Ophthalmol Vis Sci 55:881–892. https://doi.org/10.1167/iovs.13-13296
3. Weleber RG, Shults WT (1981) Digoxin retinal toxicity. Clinical and electrophysiologic evaluation of a cone dysfunction syndrome. Arch Ophthalmol 99:1568–1572. https://doi.org/10.1001/archopht.1981.03930020442007
4. Capellan P, Mahrous MA, Kovacs KD (2021) OCT findings in presumed digoxin retinal toxicity. Ophthalmology Retina 5:1173–1175. https://doi.org/10.1016/j.oret.2021.04.019
5. Georgiou M, Fujinami K, Vincent A, Nasser F, Khateb S, Vargas ME et al (2021) KCNV2-associated retinopathy: detailed retinal phenotype and structural endpoints–KCNV2 study group report 2. Am J Ophthalmol 230:1–11. https://doi.org/10.1016/j.ajo.2021.03.004
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3. Digoxin;Reactions Weekly;2023-10-28
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