Differential effects of Bcl-2 and caspases on mitochondrial permeabilization during endogenous or exogenous reactive oxygen species-induced cell death-Reference-Cited by-同舟云学术

Differential effects of Bcl-2 and caspases on mitochondrial permeabilization during endogenous or exogenous reactive oxygen species-induced cell death

Published:2012-04-11 Issue:4 Volume:28 Page:239-253
ISSN:0742-2091
Container-title:Cell Biology and Toxicology
language:en
Short-container-title:Cell Biol Toxicol

Author:

Rincheval Vincent,Bergeaud Marie,Mathieu Lise,Leroy Jacqueline,Guillaume Arnaud,Mignotte Bernard,Le Floch Nathalie,Vayssière Jean-Luc

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Link

http://link.springer.com/content/pdf/10.1007/s10565-012-9219-9.pdf

Reference21 articles.

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4. Dive C, Gregory CD, Phipps DJ, Evans DL, Milner AE, Wyllie AH. Analysis and discrimination of necrosis and apoptosis (programmed cell death) by multiparameter flow cytometry. Biochim Biophys Acta. 1992;1133(3):275–85.

5. Dumay A, Rincheval V, Trotot P, Mignotte B, Vayssiere JL. The superoxide dismutase inhibitor diethyldithiocarbamate has antagonistic effects on apoptosis by triggering both cytochrome c release and caspase inhibition. Free Radic Biol Med. 2006;40(8):1377–90.

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