Short-term vitamin A supplementation at therapeutic doses induces a pro-oxidative state in the hepatic environment and facilitates calcium-ion-induced oxidative stress in rat liver mitochondria independently from permeability transition pore formation-Reference-Cited by-同舟云学术

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Short-term vitamin A supplementation at therapeutic doses induces a pro-oxidative state in the hepatic environment and facilitates calcium-ion-induced oxidative stress in rat liver mitochondria independently from permeability transition pore formation

Published:2008-11-29 Issue:6 Volume:25 Page:545-560
ISSN:0742-2091
Container-title:Cell Biology and Toxicology
language:en
Short-container-title:Cell Biol Toxicol

Author:

de Oliveira Marcos Roberto,Oliveira Max William Soares,Lorenzi Rodrigo,Fagundes da Rocha Ricardo,Fonseca Moreira José Cláudio

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Link

http://link.springer.com/content/pdf/10.1007/s10565-008-9111-9.pdf

Reference54 articles.

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2. Aon MA, Cortassa S, Maack C, O’Rourke B. Sequential opening of mitochondrial ion channels as a function of glutathione redox thiol status. J Biol Chem 2007;282:21889–900. doi: 10.1074/jbc.M702841200 .

3. Badger DA, Sauer JM, Hoglen NC, Jolley CS, Sipes IG. The role of inflammatory cells and cytochrome P450 in the potentiation of CCl4-induced liver injury by a single dose of retinol. Toxicol Appl Pharmacol 1996;141:507–19. doi: 10.1006/taap.1996.0316 .

4. Borutaite V, Morkuniene R, Brown GC. Release of cytochrome c from heart mitochondria is induced by high Ca2+ and peroxynitrite and is responsible for Ca2+-induced inhibition of substrate oxidation. Biochim Biophys Acta 1999;1453:41–8.

5. Cai J, Jones DP. Superoxide in apoptosis. Mitochondrial generation triggered by citochrome c loss. J Biol Chem 1998;273:11401–4. doi: 10.1074/jbc.273.19.11401 .

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