Human umbilical cord blood–derived MSCs exosome attenuate myocardial injury by inhibiting ferroptosis in acute myocardial infarction mice-Reference-Cited by-同舟云学术

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Human umbilical cord blood–derived MSCs exosome attenuate myocardial injury by inhibiting ferroptosis in acute myocardial infarction mice

Published:2020-06-13 Issue:1 Volume:37 Page:51-64
ISSN:0742-2091
Container-title:Cell Biology and Toxicology
language:en
Short-container-title:Cell Biol Toxicol

Author:

Song Yufang,Wang Baocai,Zhu Xiliang,Hu Junlong,Sun Junjie,Xuan Jizhong,Ge Zhenwei

Funder

The Key Science and Technology Research Projects in Henan Province

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Link

https://link.springer.com/content/pdf/10.1007/s10565-020-09530-8.pdf

Reference22 articles.

1. Baba Y, Higa JK, Shimada BK, Horiuchi KM, Suhara T, Kobayashi M, et al. Protective effects of the mechanistic target of rapamycin against excess iron and ferroptosis in cardiomyocytes. Am J Physiol Heart Circ Physiol. 2018;314(3):H659–H68.

2. Bian S, Zhang L, Duan L, Wang X, Min Y, Yu H. Extracellular vesicles derived from human bone marrow mesenchymal stem cells promote angiogenesis in a rat myocardial infarction model. J Mol Med. 2014;92(4):387–97.

3. Chia-Yu W, Knutson MD. Hepatocyte divalent metal-ion transporter-1 is dispensable for hepatic iron accumulation and non-transferrin-bound iron uptake in mice. Hepatology. 2013;58(2):788–98.

4. Chipman LB, Pasquinelli AE. miRNA targeting: growing beyond the seed. Trends Genet. 2019;35(3):215–22.

5. Conrad M, Proneth B. Broken hearts: iron overload, ferroptosis and cardiomyopathy. Cell Res. 2019;29(4):263–4. https://doi.org/10.1038/s41422-019-0150-y.

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