Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption-Reference-Cited by-同舟云学术

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Mitochondria toxin-induced acute cochlear cell death indicates cellular activity-correlated energy consumption

Published:2012-11-18 Issue:9 Volume:270 Page:2403-2415
ISSN:0937-4477
Container-title:European Archives of Oto-Rhino-Laryngology
language:en
Short-container-title:Eur Arch Otorhinolaryngol

Author:

Zou Jing,Zhang Ya,Zhang Weikai,Poe Dennis,Zhai Suoqiang,Yang Shiming,Pyykkö Ilmari

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Otorhinolaryngology

Link

http://link.springer.com/content/pdf/10.1007/s00405-012-2267-1.pdf

Reference21 articles.

1. Vicente-Torres MA, Schacht J (2006) A BAD link to mitochondrial cell death in the cochlea of mice with noise-induced hearing loss. J Neurosci Res 83:1564–1572

2. Berrettini S, Forli F, Siciliano G, Mancuso M (2001) Sudden bilateral hearing loss and sporadic mitochondrial DNA deletion. J Laryngol Otol 115:128–131

3. Dehne N, Rauen U, de Groot H, Lautermann J (2002) Involvement of the mitochondrial permeability transition in gentamicin ototoxicity. Hear Res 169:47–55

4. Ohlemiller KK, Dugan LL (1999) Elevation of reactive oxygen species following ischemia-reperfusion in mouse cochlea observed in vivo. Audiol Neurootol 4:219–228

5. Hoya N, Okamoto Y, Kamiya K, Fujii M, Matsunaga T (2004) A novel animal model of acute cochlear mitochondrial dysfunction. Neuroreport 15:1597–1600

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