Interaction Between AT1 Receptor and NF-κB in Hypothalamic Paraventricular Nucleus Contributes to Oxidative Stress and Sympathoexcitation by Modulating Neurotransmitters in Heart Failure-Reference-Cited by-同舟云学术

Interaction Between AT1 Receptor and NF-κB in Hypothalamic Paraventricular Nucleus Contributes to Oxidative Stress and Sympathoexcitation by Modulating Neurotransmitters in Heart Failure

Published:2013-07-23 Issue:4 Volume:13 Page:381-390
ISSN:1530-7905
Container-title:Cardiovascular Toxicology
language:en
Short-container-title:Cardiovasc Toxicol

Author:

Yu Xiao-Jing,Suo Yu-Ping,Qi Jie,Yang Qing,Li Hui-Hua,Zhang Dong-Mei,Yi Qiu-Yue,Zhang Jian,Zhu Guo-Qing,Zhu Zhiming,Kang Yu-Ming

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1007/s12012-013-9219-x.pdf

Reference54 articles.

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2. Zhu, G. Q., Gao, L., Li, Y., Patel, K. P., Zucker, I. H., & Wang, W. (2004). AT1 receptor mRNA antisense normalizes enhanced cardiac sympathetic afferent reflex in rats with chronic heart failure. American Journal of Physiology-Heart and Circulatory Physiology, 287, H1828–H1835.

3. Francis, J., Wei, S. G., Weiss, R. M., & Felder, R. B. (2004). Brain angiotensin-converting enzyme activity and autonomic regulation in heart failure. American Journal of Physiology-Heart and Circulatory Physiology, 287, H2138–H2146.

4. Wang, H., Huang, B. S., Ganten, D., & Leenen, F. H. (2004). Prevention of sympathetic and cardiac dysfunction after myocardial infarction in transgenic rats deficient in brain angiotensinogen. Circulation Research, 94, 843–849.

5. Zhang, Z. H., Wei, S. G., Francis, J., & Felder, R. B. (2003). Cardiovascular and renal sympathetic activation by blood-borne TNF-alpha in rat: The role of central prostaglandins. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 284, R916–R927.

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