Rosuvastatin Reduces L-Type Ca2+ Current and Alters Contractile Function in Cardiac Myocytes via Modulation of β-Adrenergic Receptor Signaling
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology
Link
https://link.springer.com/content/pdf/10.1007/s12012-021-09642-5.pdf
Reference33 articles.
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3. Ozturk, N., Yaras, N., Ozmen, A., & Ozdemir, S. (2013). Long-term administration of rosuvastatin prevents contractile and electrical remodelling of diabetic rat heart. Journal of Bioenergetics and Biomembranes, 45(4), 343–352. https://doi.org/10.1007/s10863-013-9514-z.
4. Pan, Y., Li, B., Wang, J., & Li, X. (2016). Rosuvastatin alleviates type 2 diabetic atrial structural and calcium channel remodeling. Journal of Cardiovascular Pharmacology, 67(1), 57–67. https://doi.org/10.1097/FJC.0000000000000314.
5. Liu, C.-W., Yang, F., Cheng, S.-Z., Liu, Y., Wan, L.-H., & Cong, H.-L. (2017). Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore. Cardiovascular Therapeutics, 35(1), 3–9. https://doi.org/10.1111/1755-5922.12225.
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