Endoplasmic Reticulum Stress Regulates Cardiomyocyte Apoptosis in Myocardial Fibrosis Development via PERK-Mediated Autophagy-Reference-Cited by-同舟云学术

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Endoplasmic Reticulum Stress Regulates Cardiomyocyte Apoptosis in Myocardial Fibrosis Development via PERK-Mediated Autophagy

Published:2020-07-06 Issue:6 Volume:20 Page:618-626
ISSN:1530-7905
Container-title:Cardiovascular Toxicology
language:en
Short-container-title:Cardiovasc Toxicol

Author:

Zhang Changjiang,Li Yuanhong,Zhao Jingbo,Su Ke,He Ke,Da Yurong,Xia Hao

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Link

https://link.springer.com/content/pdf/10.1007/s12012-020-09586-2.pdf

Reference28 articles.

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2. Akhtar, M., Garan, H., Lehmann, M. H., & Troup, P. J. (1991). Sudden cardiac death: Management of high-risk patients. Annals of Internal Medicine, 114, 499–512.

3. Zernecke, A., & Weber, C. (2005). Inflammatory mediators in atherosclerotic vascular disease. Basic Research in Cardiology, 100, 93–101.

4. Diwan, A., Wansapura, J., Syed, F. M., Matkovich, S. J., Lorenz, J. N., & Dorn, G. W., 2nd. (2008). Nix-mediated apoptosis links myocardial fibrosis, cardiac remodeling, and hypertrophy decompensation. Circulation, 117, 396–404.

5. McDuffee, A. T., Senisterra, G., Huntley, S., Lepock, J. R., Sekhar, K. R., Meredith, M. J., et al. (1997). Proteins containing non-native disulfide bonds generated by oxidative stress can act as signals for the induction of the heat shock response. Journal of Cellular Physiology, 171, 143–151.

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