Mitochondrial Preservation in Celsior Versus Histidine Buffer Solution During Cardiac Ischemia and Reperfusion-Reference-Cited by-同舟云学术

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Mitochondrial Preservation in Celsior Versus Histidine Buffer Solution During Cardiac Ischemia and Reperfusion

Published:2009-09-15 Issue:4 Volume:9 Page:185-193
ISSN:1530-7905
Container-title:Cardiovascular Toxicology
language:en
Short-container-title:Cardiovasc Toxicol

Author:

Alves Marco G.,Oliveira Paulo J.,Carvalho Rui A.

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1007/s12012-009-9052-4.pdf

Reference33 articles.

1. Duchen, M. R. (2004). Mitochondria in health and disease: Perspectives on a new mitochondrial biology. Molecular Aspects of Medicine, 25, 365–451.

2. Crompton, M. (1999). The mitochondrial permeability transition pore and its role in cell death. Biochemical Journal, 341, 233–249.

3. Di Lisa, F., Menabo, R., Canton, M., Barile, M., & Bernardi, P. (2001). Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart. The Journal of Biological Chemistry, 276, 2571–2575.

4. Wilhelm, M. J., Pratschke, J., Laskowski, I., & Tilney, N. L. (2003). Ischemia and reperfusion injury. Transplantation Reviews, 17, 140–157.

5. Park, J. L., & Lucchesi, B. R. (1999). Mechanisms of myocardial reperfusion injury. Annals of Thoracic Surgery, 68, 1905–1912.

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