PPARγ Agonist Ameliorates the Impaired Fluidity of the Myocardial Cell Membrane and Cardiac Injury in Hypercholesterolemic Rats-Reference-Cited by-同舟云学术

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PPARγ Agonist Ameliorates the Impaired Fluidity of the Myocardial Cell Membrane and Cardiac Injury in Hypercholesterolemic Rats

Published:2015-12-17 Issue:1 Volume:17 Page:25-34
ISSN:1530-7905
Container-title:Cardiovascular Toxicology
language:en
Short-container-title:Cardiovasc Toxicol

Author:

Wu Ye,Tan Xiutao,Tian Jue,Liu Xin,Wang Yehong,Zhao Huanxin,Yan Zi,Liu Huirong,Ma Xinliang

Funder

Natural Sciences Foundation of China (NSFC)

Beijing Key Laboratory of Metabolic Disturbance Related Cardiovascular Disease Open Foundation of Capital Medical University

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1007/s12012-015-9352-9.pdf

Reference35 articles.

1. Osipov, R. M., Bianchi, C., Feng, J., Clements, R. T., Liu, Y., et al. (2009). Effect of hypercholesterolemia on myocardial necrosis and apoptosis in the setting of ischemia-reperfusion. Circulation, 120, S22–S30.

2. Cai, H., & Harrison, D. G. (2000). Endothelial dysfunction in cardiovascular diseases: The role of oxidant stress. Circulation Research, 87, 840–844.

3. Wang, T. D., Chen, W. J., Su, S. S., Lo, S. C., Lin, W. W., et al. (2002). Increased cardiomyocyte apoptosis following ischemia and reperfusion in diet-induced hypercholesterolemia: Relation to Bcl-2 and Bax proteins and caspase-3 activity. Lipids, 37, 385–394.

4. Karbiner, M. S., Sierra, L., Minahk, C., Fonio, M. C., Bruno, M. P., et al. (2013). The role of oxidative stress in alterations of hematological parameters and inflammatory markers induced by early hypercholesterolemia. Life Sciences, 93, 503–508.

5. Joyce, S. A., MacSharry, J., Casey, P. G., Kinsella, M., Murphy, E. F., et al. (2014). Regulation of host weight gain and lipid metabolism by bacterial bile acid modification in the gut. Proceedings of the National Academy of Sciences USA, 111, 7421–7426.

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