Central Targeting of Channelrhodopsin2 by the Motif of Potassium Channel Kv2.1 Can be Altered Due to Overexpression of the Construct
Author:
Funder
Russian Science Foundation
Publisher
Springer Science and Business Media LLC
Subject
Biomedical Engineering,Bioengineering
Link
https://link.springer.com/content/pdf/10.1007/s12668-021-00863-0.pdf
Reference24 articles.
1. Trimmer, J. S. (1991). Immunological identification and characterization of a delayed rectifier K+ channel polypeptide in rat brain. Proceedings of the National Academy of Sciences of the United States of America, 88(23), 10764–10768. https://doi.org/10.1073/pnas.88.23.10764.
2. Lim, S. T., Antonucci, D. E., Scannevin, R. H., & Trimmer, J. S. (2000). A Novel Targeting signal for proximal clustering of the Kv2.1 K+ channel in hippocampal neurons. Neuron, 25(2), 385–397. https://doi.org/10.1016/s0896-6273(00)80902-2.
3. Baker, C. A., Elyada, Y. M., Parra, A., & Bolton, M. M. (2016). Cellular resolution circuit mapping with temporal-focused excitation of soma-targeted channelrhodopsin. eLife, 5, e14193. https://doi.org/10.7554/eLife.14193.
4. Simonova, N. A., Bal, N. Y., Balaban, P. M., Volgushev, M. A., & Malyshev, A. Y. (2017). Optogenetic approach to study mechanisms of heterosynaptic plasticity in cortical neurons. Zhurnal Vysshei Nervnoi Deyatelnosti Imeni I.P. Pavlova, 67(5). https://doi.org/10.7868/S0044467717050082.
5. Mahn, M., Gibor, L., Patil, P., Cohen-Kashi Malina, K., Oring, S., Printz, Y., et al. (2018). High-efficiency optogenetic silencing with soma-targeted anion-conducting channelrhodopsins. Nature Communications, 9(1), 4125. https://doi.org/10.1038/s41467-018-06511-8.
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