Lp-PLA2 as a Marker of Cardiovascular Diseases
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11883-010-0095-6.pdf
Reference20 articles.
1. Kolodgie FD, Burke AP, Skorija KS et al.: Lipoprotein-associated phospholipase A2 protein expression in the natural progression of human coronary atherosclerosis. Arterioscler Thromb Vasc Biol 2006, 26:2523–2529.
2. Carpenter KL, Dennis IF, Challis IR, et al.: Inhibition of lipoprotein-associated phospholipase A2 diminishes the death-inducing effects of oxidised LDL on human monocyte-macrophages. FEBS Lett 2001, 505:357–363.
3. Quarck R, De Geest B, Stengel D, et al.: Adenovirus-mediated gene transfer of human platelet-activating factor-acetylhydrolase prevents injury-induced neointima formation and reduces spontaneous atherosclerosis in apolipoprotein E–deficient mice. Circulation 2001, 103:2495–2500.
4. • Tsimikas S, Tsironis LD, Tselepis AD: New insights into the role of lipoprotein(a)-associated lipoprotein-associated phospholipase A2 in atherosclerosis and cardiovascular disease. Arterioscler Thromb Vasc Biol 2007, 27:2094–2099. This article outlines the key role of Lp-PLA 2 in initiation and progression of atherosclerosis and CAD.
5. Chen CH: Platelet-activating factor acetylhydrolase: is it good or bad for you? Curr Opin Lipidol 2004, 15:337–341.
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