Fetal Membrane Organ-On-Chip: An Innovative Approach to Study Cellular Interactions
Author:
Publisher
Springer Science and Business Media LLC
Subject
Obstetrics and Gynaecology
Link
https://link.springer.com/content/pdf/10.1007/s43032-020-00184-9.pdf
Reference25 articles.
1. Behnia F, Taylor BD, Woodson M, et al. Chorioamniotic membrane senescence: a signal for parturition? Am J Obstet Gynecol. 2015;213(3):359.e1-416.e1.
2. Bredeson S, Papaconstantinou J, Deford JH, et al. HMGB1 promotes a p38MAPK associated non-infectious inflammatory response pathway in human fetal membranes. PLoS One. 2014; 9(12):e113799.
3. Dixon CL, Richardson L, Sheller-Miller S, Saade G, Menon R. A distinct mechanism of senescence activation in amnion epithelial cells by infection, inflammation, and oxidative stress. Am J Reprod Immunol. 2017;79(3).
4. Menon R, Boldogh I, Urrabaz-Garza R, et al. Senescence of primary amniotic cells via oxidative DNA damage. PLoS One. 2013;8(12):e83416.
5. Menon R, Boldogh I, Hawkins HK, et al. Histological evidence of oxidative stress and premature senescence in preterm premature rupture of the human fetal membranes recapitulated in vitro. Am J Pathol. 2014;184(6):1740–51.
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