The Mechanism of IL-17 Regulating Neutrophils Participating in Host Immunity of RVVC Mice
Author:
Publisher
Springer Science and Business Media LLC
Subject
Obstetrics and Gynecology
Link
https://link.springer.com/content/pdf/10.1007/s43032-023-01291-z.pdf
Reference32 articles.
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2. Yano J, Peters BM, Noverr MC, Fidel PL Jr. Novel mechanism behind the immunopathogenesis of vulvovaginal candidiasis: “neutrophil anergy.” Infect Immun. 2018;86(3):e00684-17. https://doi.org/10.1128/IAI.00684-17.
3. Ardizzoni A, Wheeler RT, Pericolini E. It Takes Two to Tango: How a Dysregulation of the Innate Immunity, Coupled With Candida Virulence, Triggers VVC Onset. Front Microbiol. 2021;12:692491.
4. Kalia N, Singh J, Sharma S, Kaur M. SNPs in 3'-UTR region of MBL2 increases susceptibility to recurrent vulvovaginal infections by altering sMBL levels. Immunobiology. 2019;224(1):42–9.
5. Shao MK, Qi WJ, Hou MY, Luo DD. Analysis of pathogenic factors of Candida albicans and the effect of vaginal immunization on recurrent vulvovaginal candidiasis in mice. J Obstet Gynaecol Res. 2022;48(3):857–65.
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