Association of IL-12B gene rs6887695 polymorphism with hereditary susceptibility and clinical characterization of psoriasis vulgaris in the Chinese Han population
Author:
Publisher
Springer Science and Business Media LLC
Subject
Dermatology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00403-013-1342-9.pdf
Reference24 articles.
1. Allen MH, Ameen H, Veal C et al (2005) The major psoriasis susceptibility locus PSORS1 is not a risk factor for late-onset psoriasis. J Invest Dermatol 124(1):103–106
2. Arican O, Aral M, Sasmaz S, Ciragil P (2005) Serum levels of TNF-alpha, IFN-gamma, IL-6, IL-8, IL-12, IL-17, and IL-18 in patients with active psoriasis and correlation with disease severity. Mediators Inflamm 5:273–279
3. Barker JN (1991) The pathophysiology of psoriasis. Lancet 338(8761):227–230
4. Campalani E, Allen MH, Fairhurst D et al (2006) Apolipoprotein E gene polymorphisms are associated with psoriasis but do not determine disease response to acitretin. Br J Dermatol 154(2):345–352
5. Capon F, di Meglio P, Szaub J et al (2007) Sequence variants in the genes for the interleukin-23 receptor (IL23R) and its ligand (IL12B) confer protection against psoriasis. Hum Genet 122(2):201–206
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1. Break on through: The role of innate immunity and barrier defence in atopic dermatitis and psoriasis;Skin Health and Disease;2022-02-16
2. Association of the novel susceptible locus rs9266150 with clinical features of psoriasis vulgaris in the Chinese Han population;Experimental Dermatology;2018-06-28
3. Correlation analysis of the HLA-DPB1*05:01 and BTNL2 genes within the histocompatibility complex region with a clinical phenotype of psoriasis vulgaris in the Chinese Han population;The Journal of Gene Medicine;2017-09
4. Interleukin (IL)-18, cooperatively with IL-23, induces prominent inflammation and enhances psoriasis-like epidermal hyperplasia;Archives of Dermatological Research;2017-03-15
5. A Genetic Variant rs1020760atNFKB1is Associated with Clinical Features of Psoriasis Vulgaris in a Han Chinese Population;Annals of Human Genetics;2016-06-27
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