Signatures of epigenetic, biological and mitotic age acceleration and telomere shortening are associated with arsenic-induced skin lesions-Reference-Cited by-同舟云学术

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Signatures of epigenetic, biological and mitotic age acceleration and telomere shortening are associated with arsenic-induced skin lesions

Published:2024-05-22 Issue:5 Volume:316 Page:
ISSN:1432-069X
Container-title:Archives of Dermatological Research
language:en
Short-container-title:Arch Dermatol Res

Author:

Jeremian Richie^ORCID,Lytvyn Yuliya^ORCID,Fotovati Rayyan,Li Kaiyang^ORCID,Sachdeva Muskaan^ORCID,Tarafdar Nawar,Georgakopoulos Jorge R.,Piguet Vincent^ORCID,Litvinov Ivan V.^ORCID,Yeung Jensen^ORCID

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s00403-024-02923-3.pdf

Reference15 articles.

1. Huang HW, Lee CH, Yu HS (2019) Arsenic-induced carcinogenesis and immune dysregulation. Int J Environ Res Public Health 16(15):2746

2. Al-Eryani L, Jenkins SF, States VA, Pan J, Malone JC, Rai SN et al (2018) miRNA expression profiles of premalignant and malignant arsenic-induced skin lesions. PLoS ONE 13(8):e0202579

3. Guo X, Chen X, Wang J, Liu Z, Gaile D, Wu H et al (2018) Multi-generational impacts of arsenic exposure on genome-wide DNA methylation and the implications for arsenic-induced skin lesions. Environ Int 119:250–263

4. Chen L, Ganz PA, Sehl ME (2022) DNA methylation, aging, and cancer risk: a mini-review. Front Bioinform 2:847629

5. Horvath S (2013) DNA methylation age of human tissues and cell types. Genome Biol 14(10):R115

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