A possible implication of reduced levels of LIF, LIFR, and gp130 in vasculopathy related to systemic sclerosis-Reference-Cited by-同舟云学术

A possible implication of reduced levels of LIF, LIFR, and gp130 in vasculopathy related to systemic sclerosis

Published:2017-10-16 Issue:10 Volume:309 Page:833-842
ISSN:0340-3696
Container-title:Archives of Dermatological Research
language:en
Short-container-title:Arch Dermatol Res

Author:

Taniguchi Takashi,Miyagawa Takuya,Tamaki Zenshiro,Nakamura Kouki,Yamashita Takashi,Saigusa Ryosuke,Takahashi Takehiro,Toyama Tetsuo,Ichimura Yohei,Yoshizaki Ayumi,Tada Yayoi,Sugaya Makoto,Kadono Takafumi,Sato Shinichi,Asano Yoshihide^ORCID

Funder

the Ministry of Health, Labour, and Welfare of Japan

Publisher

Springer Science and Business Media LLC

Subject

Dermatology,General Medicine

Link

http://link.springer.com/article/10.1007/s00403-017-1786-4/fulltext.html

Reference52 articles.

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2. Akamata K, Asano Y, Taniguchi T et al (2015) Increased expression of chemerin in endothelial cells due to Fli1 deficiency may contribute to the development of digital ulcers in systemic sclerosis. Rheumatology (Oxford) 54:1308–1316

3. Asano Y, Czuwara J, Trojanowska M (2007) Transforming growth factor-β regulates DNA binding activity of transcription factor Fli1 by p300/CREB-binding protein-associated factor-dependent acetylation. J Biol Chem 282:34672–34683

4. Asano Y (2010) Future treatments in systemic sclerosis. J Dermatol 37:54–70

5. Asano Y, Stawski L, Hant F et al (2010) Endothelial Fli1 deficiency impairs vascular homeostasis: a role in scleroderma vasculopathy. Am J Pathol 176:1983–1998

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