Hyperoxaluric rats do not exhibit alterations in renal expression patterns of Slc26a1 (SAT1) mRNA or protein
Author:
Publisher
Springer Science and Business Media LLC
Subject
Urology
Link
http://link.springer.com/content/pdf/10.1007/s00240-012-0480-4.pdf
Reference36 articles.
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2. Danpure CJ (2005) Molecular etiology of primary hyperoxaluria type 1: new directions for treatment. Am J Nephrol 25:303–310
3. Hatch M, Freel RW (2005) Intestinal transport of an obdurate anion: oxalate. Urol Res 33:1–16
4. Knight TF, Sansom SC, Senekjian HO, Weinman EJ (1981) Oxalate secretion in the rat proximal tubule. Am J Physiol 240:F295–F298
5. Senekjian HO, Weinman EJ (1982) Oxalate transport by proximal tubule of the rabbit kidney. Am J Physiol 243:F271–F275
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1. Impairment of neuro-renal cells on exposure to cosmopolitan polluted river water followed by differential protection of Launea taraxacifolia in male rats;Comparative Clinical Pathology;2019-01-16
2. Absence of the sulfate transporter SAT-1 has no impact on oxalate handling by mouse intestine and does not cause hyperoxaluria or hyperoxalemia;American Journal of Physiology-Gastrointestinal and Liver Physiology;2019-01-01
3. Sex-independent expression of chloride/formate exchanger Cfex (Slc26a6) in rat pancreas, small intestine, and liver, and male-dominant expression in kidneys;Archives of Industrial Hygiene and Toxicology;2018-12-01
4. Changes of Klotho protein and Klotho mRNA expression in a hydroxy-L-proline induced hyperoxaluric rat model;Journal of Veterinary Medical Science;2017
5. Extracellular Cl− regulates human SO4 2−/anion exchanger SLC26A1 by altering pH sensitivity of anion transport;Pflügers Archiv - European Journal of Physiology;2016-04-29
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