Erythrocyte ghost (Na+ + K+) ATPase activity in mice with hereditary muscular dystrophy (strain C57 BL 6 J/dy)
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Neurology
Link
http://link.springer.com/content/pdf/10.1007/BF00312959.pdf
Reference49 articles.
1. Araki, S., Mawatari, S.: Ouabain and erythrocyte-ghost adenosine triphosphatase: effects in human muscular dystrophies. Arch. Neurol. 24, 187?190 (1971)
2. Baker, N., Bloom, A., Blahd, W. H.: Concentrations of K and N in skeletal muscle of mice with a hereditary myopathy. Am. J. Physiol. 193, 530?533 (1958)
3. Bosia, A., Pescarmona, G. R., Arese, P.: Red cell glycolysis in the myodystrophic child. Europ. J. Clin. Invest. 1, 413?420 (1971)
4. Brown, H. D., Chattopadhyay, S. K., Patel, A., Rigdon, R. H.: Glycoside effect upon membrane enzymes in erythrocytes and muscle in Duck myopathy. Experientia 23, 522?523 (1967a)
5. Brown, H. D., Chattopadhyay, S. K., Patel, A. B.: Erythrocyte abnormality in human myopathy. Science 157, 1577?1578 (1967b)
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1. Role of intracellular calcium in promoting muscle damage: A strategy for controlling the dystrophic condition;Experientia;1978-12
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