Selective inhibition of PKCβ2 improves Caveolin-3/eNOS signaling and attenuates lipopolysaccharide-induced injury by inhibiting autophagy in H9C2 cardiomyocytes
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,General Medicine,Physiology,Histology
Link
https://link.springer.com/content/pdf/10.1007/s10735-021-09990-0.pdf
Reference45 articles.
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2. Boncoeur E, Bouvet GF, Migneault F et al (2013) Induction of nitric oxide synthase expression by lipopolysaccharide is mediated by calcium-dependent PKCalpha-beta1 in alveolar epithelial cells. Am J Physiol Lung Cell Mol Physiol 305:L175-184. https://doi.org/10.1152/ajplung.00295.2012
3. Campostrini G, Bonzanni M, Lissoni A et al (2017) The expression of the rare caveolin-3 variant T78M alters cardiac ion channels function and membrane excitability. Cardiovasc Res 113:1256–1265. https://doi.org/10.1093/cvr/cvx122
4. Chen L, Liu P, Feng X, Ma C (2017) Salidroside suppressing LPS-induced myocardial injury by inhibiting ROS-mediated PI3K/Akt/mTOR pathway in vitro and in vivo. J Cell Mol Med 21:3178–3189. https://doi.org/10.1111/jcmm.12871
5. Corsini E, Galbiati V, Esser PR et al (2014) Role of PKC-beta in chemical allergen-induced CD86 expression and IL-8 release in THP-1 cells. Arch Toxicol 88:415–424. https://doi.org/10.1007/s00204-013-1144-z
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