Amyloid-β, vitamin D: why we should triangulate conclusions about therapeutic targets in Alzheimer’s disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Psychiatry and Mental health,Neurology (clinical),Dermatology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10072-023-06840-7.pdf
Reference5 articles.
1. Munafò MR, Davey SG (2018) Robust research needs many lines of evidence. Nature 553(7689):399–401. https://doi.org/10.1038/d41586-018-01023-3
2. Daly T et al (2020) Amyloid-β in Alzheimer’s disease: a study of citation practices of the amyloid cascade hypothesis between 1992 and 2019. J Alzheimers Dis 74(4):1309–1317. https://doi.org/10.3233/JAD-191321
3. Gregory S, Saunders S, Ritchie CW (2022) Science disconnected: the translational gap between basic science, clinical trials, and patient care in Alzheimer’s disease. Lancet Healthy Longev 3(11):e797–e803. https://doi.org/10.1016/S2666-7568(22)00219-7
4. Lai RH et al (2022) Vitamin D supplementation worsens Alzheimer’s progression: animal model and human cohort studies. Aging Cell 21(8):e13670. https://doi.org/10.1111/acel.13670
5. Cummings J, Feldman HH, Scheltens P (2019) The, “rights” of precision drug development for Alzheimer’s disease. Alzheimers Res Ther 11(1):76. https://doi.org/10.1186/s13195-019-0529-5
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1. Improving Clinical Trials of Antioxidants in Alzheimer’s Disease;Journal of Alzheimer's Disease;2023-09-28
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