InsP3R–RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle-Reference-Cited by-同舟云学术

InsP3R–RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle

Published:2022-11-15 Issue:1 Volume:117 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Jin Xin,Amoni Matthew,Gilbert Guillaume,Dries Eef,Doñate Puertas Rosa,Tomar Ashutosh,Nagaraju Chandan K.,Pradhan Ankit,Yule David I.,Martens Tobie,Menten Roxane,Vanden Berghe Pieter,Rega Filip,Sipido Karin,Roderick H. Llewelyn^ORCID

Funder

Fonds Wetenschappelijk Onderzoek

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

https://link.springer.com/content/pdf/10.1007/s00395-022-00967-y.pdf

Reference63 articles.

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2. Bare DJ, Kettlun CS, Liang M, Bers DM, Mignery GA (2005) Cardiac type 2 inositol 1,4,5-trisphosphate receptor: interaction and modulation by calcium/calmodulin-dependent protein kinase II. J Biol Chem 280:15912–15920. https://doi.org/10.1074/jbc.M414212200

3. Bers DM (2002) Cardiac excitation-contraction coupling. Nature 415:198–205. https://doi.org/10.1038/415198a

4. Bers DM, Pogwizd SM, Schlotthauer K (2002) Upregulated Na/Ca exchange is involved in both contractile dysfunction and arrhythmogenesis in heart failure. Basic Res Cardiol 97(Suppl 1):I36-42. https://doi.org/10.1007/s003950200027

5. Betzenhauser MJ, Wagner LE 2nd, Iwai M, Michikawa T, Mikoshiba K, Yule DI (2008) ATP modulation of Ca2+ release by type-2 and type-3 inositol (1, 4, 5)-triphosphate receptors. Differing ATP sensitivities and molecular determinants of action. J Biol Chem 283:21579–21587. https://doi.org/10.1074/jbc.M801680200

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