Inhibition of cardiac CaMKII to cure heart failure: step by step towards translation?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s00395-016-0582-1.pdf
Reference45 articles.
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2. Anderson ME (2015) Oxidant stress promotes disease by activating CaMKII. J Mol Cell Cardiol 89:160–167. doi: 10.1016/j.yjmcc.2015.10.014
3. Backs J, Song K, Bezprozvannaya S, Chang S, Olson EN (2006) CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy. J Clin Invest 116:1853–1864. doi: 10.1172/JCI27438
4. Bell JR, Raaijmakers AJA, Curl CL, Reichelt ME, Harding TW, Bei A, Ng DCH, Erickson JR, Petroff MV, Harrap SB, Delbridge LMD (2015) Cardiac CaMKIIδ splice variants exhibit target signaling specificity and confer sex-selective arrhythmogenic actions in the ischemic-reperfused heart. Int J Cardiol 181:288–296. doi: 10.1016/j.ijcard.2014.11.159
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